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GBZ50-2015 English PDF

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GBZ50-2015: Diagnosis of occupational acrylamide poisoning
Status: Valid

GBZ50: Evolution and historical versions

Standard ID	Contents [version]	USD	STEP2	[PDF] delivered in	Standard Title (Description)	Status	PDF
GBZ 50-2015	English	129	Add to Cart	3 days [Need to translate]	Diagnosis of occupational acrylamide poisoning	Valid	GBZ 50-2015
GBZ 50-2002	English	279	Add to Cart	3 days [Need to translate]	Diagnostic Criteria of Occupational Chronic Acrylamide Poisoning	Obsolete	GBZ 50-2002

PDF similar to GBZ50-2015

Standard similar to GBZ50-2015

GBZ 57 GBZ 20 GBZ 49 GBZ 59 GBZ 40

Basic data

Standard ID	GBZ 50-2015 (GBZ50-2015)
Description (Translated English)	Diagnosis of occupational acrylamide poisoning
Sector / Industry	National Standard
Classification of Chinese Standard	C60
Classification of International Standard	13.1
Word Count Estimation	6,688
Date of Issue	2015-04-21
Date of Implementation	2015-11-01
Older Standard (superseded by this standard)	GBZ 50-2002
Quoted Standard	GB/T 16180; GBZ 18; GBZ 20; GBZ 76; GBZ/T 247; GBZ/T 228
Regulation (derived from)	State-Health-Communication 2015 No.6
Issuing agency(ies)	National Health and Family Planning Commission
Summary	This Standard specifies the principles of diagnosis of occupational acute and chronic acrylamide poisoning, diagnostic classification and treatment principles. This Standard applies to the diagnosis and treatment of occupational acute and chronic acrylamide poisoning.

GBZ50-2002: Diagnostic Criteria of Occupational Chronic Acrylamide Poisoning

---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Chronic Acrylamide Poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational chronic acrylamide poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China

Foreword

Article 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 16370-1996 inconsistent with this standard, subject to this standard. In the long-term exposure to acrylamide in the professional activities, can occur chronic acrylamide poisoning. To protect the body of the body Kang, effective prevention and treatment of chronic acrylamide poisoning, has released GB 16370-1996. This standard highlights the damage to the nervous system by acrylamide and is classified according to its degree of damage. Appendix A to this standard is an informative appendix and Appendix B is a normative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. This standard by the Chinese Center for Disease Control and Prevention of Occupational Health and Poison Control and Shandong Qilu Petrochemical Company Responsible for drafting, Zhejiang Academy of Medical Sciences and Heilongjiang Province Institute of Labor and Occupational Health to participate in the drafting. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational chronic acrylamide poisoning Occupational chronic acrylamide poisoning is the production and use of the process due to close contact with acrylamide due to nervous system change Become the main disease.

1 Scope

This standard specifies the diagnostic criteria and principles of occupational chronic acrylamide poisoning. This standard applies to the diagnosis and treatment of occupational chronic acrylamide poisoning.

2 normative reference documents

The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest The version applies to this standard. GBZ 76 Occupational acute chemical poisoning Nervous system disease diagnostic criteria

3 diagnostic principles

According to the occupational history of exposure to acrylamide, with multiple peripheral nerve damage and neuromuscular changes or cerebellum Symptoms of symptoms, signs, combined with on-site hygiene investigation, and exclude other causes of similar diseases, can be diagnosed.

4 observation object

Any of the following, can be listed as the object of observation; a) exposure to acrylamide local skin appear sweating, wet, peeling, erythema; b) appear numbness, tingling, lower limb fatigue, lethargy and other symptoms; c) Neuro-electromyography shows suspicious neurogenic damage.

5 Diagnostic and grading standards

5.1 mild poisoning Have any of the first two items of observation, at the same time have any of the following, can be diagnosed with mild poisoning. a) limb distal tuning fork vibration or pain, touch disorders, accompanied by achilles tendon reflex; b) bilateral Achilles tendon reflex; c) Neuro-electromyography shows neurogenic damage. 5.2 moderate poisoning In the case of mild poisoning, with any of the following, can be diagnosed as moderate poisoning. a) limb tremor or pain, touch the level of elbow, knee above, accompanied by Achilles tendon reflex; b) sensory ataxia; c) electromyography showed neurogenic damage, and there are more spontaneous nerve potential. 5.3 severe poisoning Those who have any of the following can be diagnosed with severe poisoning. a) significant drowsiness and cerebellar dysfunction; b) distal limb hypertrophy, and affect motor function.

6 Principles of handling

6.1 Principles of treatment Can be used B vitamins, energy mixture, and supplemented by physical therapy, physical therapy and symptomatic treatment. Severe poisoning should be strengthened at the same time Supportive therapy. 6.2 Other treatments 6.2.1 Observe objects Generally do not leave the acrylamide operation, half a year review, as much as possible nerve - EMG examination, dynamic observation. 6.2.2 mild poisoning Temporarily removed from the period of exposure to acrylamide during the sick, after treatment can be restored to the original work, and regular review. 6.2.3 moderate and severe poisoning Should be transferred from acrylamide and other harmful to the nervous system operations, after treatment according to the results of the arrangements for rest or work For.

7 Correctly use the instructions in this standard

See Appendix A (informative), Appendix B (normative).

Appendix A

(Informative) Correctly use the instructions in this standard A.1 This standard applies to persons engaged in the production or use of acrylamide monomer operations, such as the production of polyacrylamide, N, N- Methylene bisacrylamide, N-methylol acrylamide and the like. A.2 Skin contact is the main route of occupational acrylamide poisoning, so the close contact with acrylamide is mainly Refers to the degree of skin contamination, followed by the workshop air concentration. A.3 mild to moderate acrylamide poisoning to peripheral nerve damage as the main performance, the two grading limit is moderate poisoning, The range of sensory disturbances is extended to elbow, knee level, or deep sensory disturbances caused by ataxia; when there is limb distal muscle Was atrophic, and affect the motor function or cerebellar dysfunction (either for the first performance or in the existing peripheral neuropathy On the emergence of those who should be diagnosed as severe poisoning. A.4 long-term exposure to low concentrations of acrylamide, the main manifestations of chronic occult disease in the incidence of peripheral neuropathy. Short-term access After exposure to high concentrations of acrylamide can occur in a month or so cerebellar dysfunction, the incidence of faster, can also refer to this standard. A.5 severe poisoning of cerebellar dysfunction in the contact after a few weeks can subside, followed by peripheral nerve damage. A.6 limb tremor disorder and Achilles tendon reflex is the early manifestation of mild poisoning, therefore, must be repeated carefully check the two Subject signs, check the Achilles tendon reflex should take the prone knees or refer to the reinforcement method. A.7 deep sensation (vibration sensation, position sensation) disorder caused by ataxia, mainly for the feet can not be in a straight line Walking, single foot can not stand and close your eyes difficult. Cerebellar dysfunction, including eye-level tremor, verbal ambiguity Was painful, lower limb muscle tension, nasal and knee tendon test instability, rotation action disorders, gait staggering. A.8 nerve - electromyography on the early diagnosis of this disease is important. Acrylamide poisoning is caused by peripheral nerve axon damage Therefore, should focus on checking the distal muscles of the limbs of the EMG and sensory nerve potential. Inspection method and its result judgment benchmark See GBZ 76. A.9 need to rule out the cause of peripheral neuropathy and cerebellar ataxia of various diseases, such as furan, isoniazid, arsenic, disulfide Carbonized, chloropropene, methyl n-butyl ketone, n-hexane and other poisoning and diabetes, infectious multiple neuritis and other diseases.

Appendix B

(Normative appendix) Neurological - electromyographic examination method and its judgment of neurogenic damage See GBZ 76.

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