GBZ63: Evolution and historical versions
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| GBZ 63-2017 | English | RFQ |
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Diagnostic Criteria of Occupational Acute Barium Poisoning
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GBZ 63-2017
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| GBZ 63-2002 | English | 199 |
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Diagnostic Criteria of Occupational Acute Barium Poisoning
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GBZ 63-2002
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PDF similar to GBZ63-2017
Standard similar to GBZ63-2017 GBZ 20 GBZ 57 GBZ 49 GBZ 59 GBZ 68 Basic data | Standard ID | GBZ 63-2017 (GBZ63-2017) | | Description (Translated English) | Diagnostic Criteria of Occupational Acute Barium Poisoning | | Sector / Industry | National Standard | | Classification of Chinese Standard | C60 | | Date of Issue | 2017-05-18 | | Date of Implementation | 2017-11-01 | | Older Standard (superseded by this standard) | GBZ 63-2002 | | Regulation (derived from) | State-Health-Communication (2017) 5 | | Issuing agency(ies) | General Administration of Quality Supervision, Inspection and Quarantine of the People's Republic of China, Standardization Administration of the People's Republic of China | GBZ63-2002: Diagnostic Criteria of Occupational Acute Barium Poisoning---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Acute Barium Poisoning
ICS 13.100
C60
GBZ
People's Republic of China National Occupational Health Standards
Diagnostic criteria for occupational acute barium poisoning
Released in.2002-04-08
2002-06-01 Implementation
Issued by the Ministry of Health of the People's Republic of China
Foreword
Article 6.1 of this standard is recommended and the remainder is mandatory.
According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard
GB 16384-1996 inconsistent with this standard, the standard shall prevail.
Barium compounds for the chemical raw materials, in the production, transportation, use the process, such as improper protection, short-term contact with a large number of soluble
Barium compounds can cause muscle palsy, cardiovascular damage and hypokalemia-based acute poisoning, and may have early symptoms
Obviously sudden death occurred. For the benefit of early diagnosis and treatment of poisoning to protect the health of workers in the summary
Domestic clinical practice experience and analysis of domestic and foreign research on the basis of the development of this standard.
Appendix A to this standard is an informative appendix.
This standard is proposed and centralized by the Ministry of Health of the People's Republic of China.
This standard is drafted by the Second Affiliated Hospital of Shanxi Medical University.
This standard is interpreted by the Ministry of Health of the People's Republic of China.
Diagnostic criteria for occupational acute barium poisoning
Occupational acute barium poisoning refers to the production, transportation and use of the process, a short time exposure to a large number of soluble barium compounds
Caused by muscle paralysis, cardiovascular damage and hypokalemia as the main manifestation of systemic diseases.
1 Scope
This standard specifies the diagnostic criteria and principles of occupational acute barium poisoning.
This standard applies to the production of labor and other professional activities, a large number of barium compounds through the respiratory tract, digestive tract, burns
Or damaged skin into the body caused by occupational acute poisoning. Does not apply to long-term exposure to low concentrations of barium compounds caused
Other diseases.
2 normative reference documents
The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document
All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however,
The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest
The version applies to this standard.
GBZ 76 Occupational acute chemical poisoning Nervous system disease diagnostic criteria
GBZ 51 Occupational chemical skin burns diagnostic criteria
3 diagnostic principles
According to the exact exposure to a large number of barium compounds in the occupational history, with muscle paralysis, cardiovascular damage, hypokalemia
Clinical manifestations, and ECG, serum potassium test results, combined with field investigation, a comprehensive analysis, excluding other reasons cited
From a similar disease, can be diagnosed.
4 contact reaction
Dizziness or headache, throat, nausea, mild abdominal pain and diarrhea and other nerve and digestive system symptoms, electrocardiogram, serum
Potassium normal, in a few hours to two days can be self-recovery.
5 Diagnostic and grading standards
5.1 mild poisoning
In addition to the above symptoms, and chest tightness, palpitations, numbness, weakness, physical weakness, strength IV level (see
GBZ 76). ECG has early hypokalemia seen or slightly lower serum potassium
5.2 moderate poisoning
Muscle strength Ⅱ ~ Ⅲ level (see GBZ 76), muscle tension decreased. ECG, serum potassium showed low potassium performance.
5.3 severe poisoning
Limbs flaccid paralysis, muscle strength 0 ~ I level (see GBZ 76), and even respiratory muscle paralysis. Electrocardiogram and serum potassium display
Significant hypokalemia, accompanied by severe arrhythmia, conduction block.
6 Principles of handling
6.1 Principles of treatment
6.1.1 immediately from the scene, skin burns with 2% to 5% sodium sulfate solution after thorough rinse and then burn conventional treatment (see
GBZ 51), barium compound dust through the respiratory tract and digestive tract entrants, mouthwash, oral sodium sulfate.
6.1.2 Respondents who respond to reactors and accidents should be closely monitored for 48 h with prophylactic treatment.
6.1.3 special effects treatment, first of all should be timely, adequate potassium, ECG and serum potassium under strict supervision, until the test
Indicators returned to normal, and then discretionary reduction, stable after the withdrawal. While intravenous or intravenous sodium sulfate or sodium thiosulfate solution.
6.1.4 Other first aid measures and symptomatic treatment are the same as internal medicine.
6.2 Other treatments
6.2.1 light and moderate poisoning after cure, can restore the original work.
6.2.2 severe poisoning should be removed after the original job.
7 Correctly use the instructions in this standard
See Appendix A (informative).
Appendix A
(Informative)
Correctly use the instructions in this standard
A.1 accidental exposure to high concentrations of barium compound dust, while entering the respiratory tract at the same time, you can enter from the digestive tract, so
Clinical manifestations and living poisoning is basically the same treatment can not be ignored when the treatment of the digestive tract.
A.2 by the skin to absorb poisoning, early symptoms of mild or lack of digestive tract, we must pay attention to ECG and serum potassium monitoring,
Beware of systemic poisoning and timely treatment, so as to avoid sudden death.
A.3 diagnostic grading standards, mainly to limb numbness, athleticism, muscle strength decreased as a starting point for the diagnosis of mild poisoning. Out
Is the limb is not complete relaxation pain paralysis, electrocardiogram, serum potassium has low potassium performance as a moderate poisoning boundaries. Mild to moderate poisoning
The change in muscle strength refers to the loss of muscle strength of two limbs or two or more limbs. Severe poisoning to limbs complete paralysis, and even
To the trunk and respiratory muscles and have severe arrhythmia, conduction block and serum potassium was significantly reduced as a major indicator.
A.4 differential diagnosis should pay attention to exclude cyclical paralysis, myasthenia gravis, progressive muscular dystrophy, peripheral neuropathy, acute
Sexual polyneuropathy (Guillain-Barre) and so on.
A.5 on the use of potassium chloride, generally for the contact reaction and mild poisoning, you can also intravenous infusion. Medium and severe
Poisoning were intravenous administration, concentration and speed according to the disease to master, avoid intravenous injection. Potassium must be in ECG monitoring and blood
Clear potassium monitoring carried out, when the condition relief, ECG, serum potassium returned to normal, the reduction of maintenance, not a sudden withdrawal to
Anti-disease repeated. The use of sodium sulfate can be used 2% sodium sulfate solution 500 mL intravenous or 10% sodium sulfate solution 20 mL
Cut the note.
A.6 When barium sulfide poisoning, in addition to the toxic effects of barium ions, can still produce hydrogen sulfide and cause the corresponding poisoning, in the diagnosis
Treatment should be noted.
A.7 high temperature barium compound burns skin poisoning, in addition to special treatment, the local burn should also be in accordance with the provisions of GBZ 51
To carry out diagnosis and treatment.
A.8 on the determination of blood barium is specific, but the examination is not yet universal, and the clinical development of poisoning and
Blood barium has nothing to do, it is not included in the standard.
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