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GBZ30-2015 English PDF

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GBZ30-2015: Diagnosis of occupational acute aromatic amino or nitro-compounds poisoning
Status: Valid

GBZ30: Evolution and historical versions

Standard ID	Contents [version]	USD	STEP2	[PDF] delivered in	Standard Title (Description)	Status	PDF
GBZ 30-2015	English	129	Add to Cart	3 days [Need to translate]	Diagnosis of occupational acute aromatic amino or nitro-compounds poisoning	Valid	GBZ 30-2015
GBZ 30-2002	English	239	Add to Cart	3 days [Need to translate]	Diagnostic Criteria of Occupational Acute Aromatic Amino-and or Nitro -- Compounds Poisoning	Obsolete	GBZ 30-2002

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Basic data

Standard ID	GBZ 30-2015 (GBZ30-2015)
Description (Translated English)	Diagnosis of occupational acute aromatic amino or nitro-compounds poisoning
Sector / Industry	National Standard
Classification of Chinese Standard	C60
Classification of International Standard	13.1
Word Count Estimation	6,615
Date of Issue	2015-09-09
Date of Implementation	2016-03-01
Older Standard (superseded by this standard)	GBZ 30-2002
Quoted Standard	GB/T 16180; GBZ 18; GBZ 51; GBZ 59; GBZ 71; GBZ 75; GBZ 79; WS/T 55; WS/T 56; WS/T 57; WS/T 58
Regulation (derived from)	State-Health-Communication 2015 No.12
Issuing agency(ies)	National Health and Family Planning Commission
Summary	This Standard specifies the amino occupational acute benzene, diagnostic criteria and principles of nitro compounds poisoning. This Standard applies to professional activities in the diagnosis and treatment of exposure to benzene, nitro compounds cause acute poisoning. Amino non-occupational acute benzene poisoning can refer to the use of nitro compounds. This Standard is not applicable to the diagnosis and treatment of occupational trinitrotoluene poisoning.

GBZ30-2002: Diagnostic Criteria of Occupational Acute Aromatic Amino-and or Nitro -- Compounds Poisoning

---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Acute Aromatic Amino-and or Nitro.Compounds Poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Occupational acute benzene amino, nitro compound poisoning diagnostic criteria Diagnostic Criteria of Occupational Acute Aromatic Amino- and Nitro-Compounds Poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China

Foreword

Article 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 8788-1988 inconsistent with this standard, the standard shall prevail. In the professional activities, short-term exposure to high concentrations of benzene amino, nitro compounds can occur acute benzene amino, nitro Base compound poisoning. In order to protect the health of the contact person, effectively control the acute benzene amino, nitro compound poisoning, and Timely diagnosis and treatment of poisoning patients, GB 8788-1988 was revised. The revised standard is in line with the occupational acute poisoning of the general standard of convergence, but also highlight the acute benzene amino, nitrate Based compound poisoning itself characteristics of the principle, according to domestic and foreign research progress in recent decades, the original standard in the diagnosis, diagnosis Broken grade and treatment principles were modified and added to make it more clear, reasonable and easy to use. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China Ministry of Health. This standard is responsible for the drafting unit. Tianjin Occupational Disease Prevention and Control Institute, Shandong Province Labor and Occupational Disease Prevention and Control Research Institute. Participated in the drafting unit. Heilongjiang Province Labor and Health Occupational Disease Prevention and Control Institute, Yantai City Occupational Disease Prevention and Control Institute, Zibo City Occupation Disease prevention and treatment hospital. This standard is interpreted by the Ministry of Health of the People's Republic of China Ministry of Health. Occupational acute benzene amino, nitro compound poisoning diagnostic criteria Occupational acute benzene amino, nitro compound poisoning is in the professional activities, short-term exposure to high concentrations of benzene amino, Nitro compounds caused by methemoglobinemia-based systemic disease, may be associated with hemolytic anemia, and liver, Kidney damage.

1 Scope

This standard specifies the occupational acute benzene amino, nitro compound poisoning diagnostic criteria and treatment principles. This standard applies to occupational activities in contact with benzene amino, nitro compounds caused by the diagnosis of acute poisoning and Reason. Non-occupational acute benzene amino, nitro compounds poisoning can refer to the use. This standard does not apply to the diagnosis and treatment of occupational trinitrotoluene poisoning.

2 normative reference documents

The terms of the following documents are hereby incorporated by reference into this standard. Any reference to the date of the document, All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to this agreement have agreed on whether the latest versions of these documents can be used. Where do not refer to the date of the reference file, Its latest version applies to this standard. GBZ 18 diagnostic criteria for occupational dermatosis (general) Diagnostic criteria for occupational toxic liver disease Diagnostic criteria for blood system diseases in occupational acute chemical poisoning Diagnostic criteria for occupational acute kidney disease Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 16180

3 diagnostic principles

According to the short-term exposure to high concentrations of benzene amino, nitro compounds in the occupational history, the emergence of methemoglobinemia The main clinical manifestations, combined with on-site hygiene survey results, comprehensive analysis, discharge of other causes caused by similar diseases, Can be diagnosed.

4 contact reaction

Exposure to benzene amino, nitro compounds with mild dizziness, headache, fatigue, chest tightness, methemoglobin below 10% in the short term can be fully restored.

5 Diagnostic and grading standards

5.1 mild poisoning Lips, auricle, tongue and finger (toe) a cyanosis, may be associated with dizziness, headache, fatigue, chest tightness, methemoglobin In 10% to 30%, usually within 24 hours to return to normal. 5.2 moderate poisoning Skin, mucous membranes were cyanosis, there may be palpitations, shortness of breath, loss of appetite, nausea, vomiting and other symptoms, high iron blood red Protein between 30% and 50%, or methemoglobin less than 30% with any of the following. a) mild hemolytic anemia, Herne body can be slightly elevated; b) chemical cystitis; c) mild liver damage; d) mild renal damage. 5.3 severe poisoning Skin and mucous membranes severe cyanosis, methemoglobin higher than 50%, and there may be disturbance of consciousness, or methemoglobin Less than 50% with any of the following. a) Herne body can be significantly increased, and secondary hemolytic anemia; b) severe toxic liver disease; c) Severely toxic nephropathy.

6 Principles of handling

6.1 Principles of treatment 6.1.1 quickly out of the scene, remove the skin pollution, immediately oxygen, close observation. 6.1.2 methemoglobinemia with hypertonic glucose, vitamin C, low-dose Meilan treatment. 6.1.3 hemolytic anemia, mainly for symptomatic and supportive care, focusing on the protection of renal function, alkalizing urine, application Appropriate amount of adrenal glucocorticoid, severe blood transfusion should be treated, if necessary, using blood transfusion therapy or blood purification therapy. Reference GBZ 75. 6.1.4 chemical cystitis, mainly alkaline urine, the application of appropriate amount of adrenal glucocorticoid, prevention and treatment of secondary infection. And can give antispasmodic agents and supportive treatment. 6.1.5 liver and kidney damage, treatment principles see GBZ 59 and GBZ 79. 6.2 Other treatments Light, moderate poisoning after cure, can restore the original work. Severe poisoning, depending on the disease recovery may be considered to withdraw from the original work For. For the identification of labor capacity, according to the relevant provisions of GB/T 16180.

7 Correctly use the instructions in this standard

See Appendix A (informative).

Appendix A

(Informative) Correctly use the instructions in this standard A.1 benzene amino, nitro compounds of many types, have a common physical and chemical properties and toxicity, but the difference is acute toxicity Large, its common toxic effect is the formation of methemoglobin, may be associated with hemolysis, liver, kidney damage. But 2-methyl-4 Nitroaniline does not appear methemoglobinemia, clinical manifestations of severe liver damage. O-toluidine, p-toluene Ammonium chloride o-toluidine in addition to the formation of methemoglobin, can cause chemical cystitis, clinical manifestations of urgency, frequent urination, Dysuria, naked eye or microscopic hematuria. A.2 benzene amino, nitro compounds poisoning, early prominent signs of cyanosis, this time other symptoms are not obvious, this The characteristics contribute to the early clinical diagnosis of these compounds poisoning. Methemoglobin is an early diagnostic indicator of poisoning. A small amount Methemoglobin formation after the natural reduction, and after appropriate treatment can be restored faster, so blood samples should be carried out in the early. Methemoglobin up to 10% of the diagnosis of the starting point of the disease, the standard classification based on the degree of methemoglobin, Hemolysis, liver damage, the degree of renal damage may be. A.3 Benzene amino, nitro compounds in the hemolysis and methemoglobin formation is very close, but not complete parallel. Nitrobenzene, o-nitrochlorobenzene, p-nitrochlorobenzene, o-nitrotoluene, the formation of methemoglobin is stronger than The formation of He En Zi body (denatured globin body) role. M-dinitrobenzene, m-nitroaniline p-nitroaniline form Hearn The role of the body is stronger than the role of the formation of methemoglobin, more prone to hemolysis, so poisoning patients in addition to the determination of high iron blood red Protein, but also the determination of erythrocyte Herne body. A.4 hemolytic diagnosis based on clinical manifestations and laboratory tests, fever (fever), headache, soy sauce, urine, jaundice, Anemia; hemoglobin and red blood cells decreased, reticulocytes increased, serum indirect bilirubin increased, urinary bladder positive, urine Occult blood positive, hemolytic period may be associated with peripheral white blood cells increased. The classification of hemolytic anemia, with reference to GBZ 80. In the disease Hehe Enzi body appears as a precursor to hemolysis, moderate poisoning Herne body is often higher than 20%, severe poisoning He En Zi body Often higher than 50%. Hearn's body peaked three to five days after poisoning. Should be closely observed after poisoning red blood cells Enzi body, hemoglobin, red blood cells, reticulocytes, serum indirect bilirubin, urinary bladder, urinary occult blood. A.5 methemoglobinemia can also be caused by drugs or other chemicals poisoning, such as the spread of malaria star, nitrite, Primary amine quinoline, potassium chlorate, bismuth sub-nitric acid, sulfonamides, phenacetin, phenyl sulfone and other causes, also found in high iron blood red Proteins, should be combined with the relevant information to be excluded. Hao Enzi appearance of the body can also be caused by other diseases, such as instability Hemoglobin disease, 6-phosphate dehydrogenase deficiency. A.6 Most of this class of poison due to the absorption of the skin and poisoning, so the timely removal of skin pollution is essential for the treatment of One of the important measures. A.7 methemoglobinemia treatment. contact reaction only need to rest, taking sugary drinks, vitamin C, if necessary With 50% glucose solution 40-60ml by adding 0.5-1.0g vitamin C intravenous injection. Mild methemoglobinemia, Can be given 1% of the United States Blue (Amy Blue) 5ml or 1mg/kg by adding 25% glucose solution 20 ~ 40ml, slow intravenous injection, Once can be. If necessary, give vitamin C. Moderate and severe methemoglobinemia, can be given 1% US blue 5-10ml Or 1-2mg/kg by adding 25% glucose solution 20-40ml, slow intravenous injection. If necessary, repeat it for 2-4 hours With one time. According to the results of dynamic determination of methemoglobin methemoglobin may be appropriate 2-4 times. At the same time can be given vitamin C and auxiliary Enzyme A and Vitamin B12. When the second dose of the United States and blue effect is not obvious, you should actively find the reasons, such as poison did not remove the dry Net, burns improperly handled, and should not be blindly repeated application. A.8 hemolytic anemia without special treatment, the treatment is focused on the protection of renal function, when the body containing Hernan erythrocyte When the proportion is greater than 50%, the exchange can be carried out as early as possible. A.9 This standard does not include benzene amino, nitro compounds caused by skin damage, such as the occurrence, can refer to GBZ 18 Reason.

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