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GBZ4-2022 English PDF

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GBZ4-2022: Diagnostic standard for occupational carbon disulfide poisoning
Status: Valid

GBZ4: Evolution and historical versions

Standard ID	Contents [version]	USD	STEP2	[PDF] delivered in	Standard Title (Description)	Status	PDF
GBZ 4-2022	English	179	Add to Cart	3 days [Need to translate]	Diagnostic standard for occupational carbon disulfide poisoning	Valid	GBZ 4-2022
GBZ 4-2002	English	279	Add to Cart	3 days [Need to translate]	Diagnostic Criteria of Occupational Chronic Carbon Disulfide Poisoning	Obsolete	GBZ 4-2002

PDF similar to GBZ4-2022

Standard similar to GBZ4-2022

GBZ 57 GBZ 20 GBZ 49 GBZ 10 GBZ 6

Basic data

Standard ID	GBZ 4-2022 (GBZ4-2022)
Description (Translated English)	Diagnostic standard for occupational carbon disulfide poisoning
Sector / Industry	National Standard
Classification of Chinese Standard	C60
Classification of International Standard	13.100
Word Count Estimation	8,863
Date of Issue	2022-03-16
Date of Implementation	2022-09-01
Issuing agency(ies)	State Administration for Market Regulation, China National Standardization Administration
Summary	This standard specifies the principles of diagnosis, diagnosis and treatment of occupational carbon disulfide poisoning. This standard applies to the diagnosis and treatment of poisoning caused by occupational exposure to carbon disulfide.

GBZ4-2002: Diagnostic Criteria of Occupational Chronic Carbon Disulfide Poisoning

---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Chronic Carbon Disulfide Poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational chronic carbon disulfide poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China

Foreword

Article 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 3233-1982 inconsistent with this standard, the standard shall prevail. In chronic occupational exposure to carbon disulfide, chronic carbon disulfide poisoning can occur. To protect the body of the body Kang, effective prevention and treatment of chronic carbon disulfide poisoning, has released GB 3233-1982. This standard is a revision. The revised standard highlights the effects of chronic exposure to carbon disulfide on the nervous system, especially the peripheral nervous system, According to the degree of damage to the classification of diagnosis. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. This standard by the Chinese Center for Disease Control and Prevention of occupational health and poisoning control is responsible for drafting, to participate in the drafting of the unit has a river West Province Labor and Health Occupational Disease Prevention and Control Research Institute, Xinxiang City, occupational disease prevention and control, Xinxiang Egret Chemical Fiber Group Co., Ltd. Heart Hospital, Jiangxi Chemical Fiber Factory, Liaoning Province, occupational disease prevention and treatment hospital, Dandong City, occupational disease prevention and treatment hospital, Dandong City Chemical Fiber Factory. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational chronic carbon disulfide poisoning Occupational chronic carbon disulfide poisoning is caused by prolonged exposure to carbon disulfide in occupational activities. Become the main systemic disease.

1 Scope

This standard specifies the diagnostic criteria and principles of occupational chronic carbon disulfide poisoning. This standard applies to the diagnosis and treatment of occupational chronic carbon disulfide poisoning.

2 normative reference documents

The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, encouragement under this standard The parties to the agreement study whether the latest versions of these documents are available. For those who do not mind the date of the reference file, the latest version This applies to this standard. Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 16180 GZB76 Occupational acute chemical poisoning Nervous system disease diagnostic criteria

3 diagnostic principles

According to the long history of close contact with carbon disulfide occupational history, with multiple peripheral neuropathy clinical, neurological - EMG changes Change or toxic encephalopathy clinical manifestations, combined with on-site hygiene survey data, and exclude other causes caused by similar diseases, Can be diagnosed.

4 observation object

Have any of the following. a) headache, dizziness, fatigue, sleep disorders, memory loss, or lower limb weakness, limb numbness and other symptoms; b) retinal micro-aneurysms appear in the fundus; c) Nerve-electromyography shows typical symptoms and signs of suspected neurogenic damage without peripheral nerve damage.

5 Diagnostic and grading standards

5.1 mild poisoning Have any of the following. a) limb symmetry gloves, socks-like distribution of pain, tactile or tuning fork vibration disturbance, while Achilles tendon reflex reduction weak; b) The signs are mild or not obvious, but the neuromyogram shows neurogenic damage. 4.2 severe poisoning Have any of the following. a) distal limb sensory disturbances, Achilles tendon reflex, with limb muscle strength decreased significantly, or limb distal muscle atrophy; Electromyography showed neurogenic damage, with nerve conduction velocity was significantly slowed or evoked potential was significantly reduced; b) toxic encephalopathy; c) Toxic psychosis.

6 Principles of handling

6.1 Principles of treatment Can be used B vitamins, energy mixture, and supplemented by physical therapy, physical therapy and symptomatic treatment. Severe poisoning should be strengthened at the same time Supportive therapy. 6.2 Other treatments 6.2.1 Observe objects Generally do not withdraw from carbon disulfide operations, should be reviewed once a half, as much as possible nerve - EMG examination, the dynamic view Check. 6.2.2 mild poisoning Mildly poisoned patients after treatment recovery, can engage in other work, and regular review. 6.2.3 severe poisoning Should be transferred from carbon disulfide and other harmful operations on the nervous system, after treatment, according to the results of the arrangements for rest or work For. Need to carry out the ability to identify workers, according to GB/T 16180 treatment.

7 Correctly use the instructions in this standard

See Appendix A (informative).

Appendix A

(Informative) Correctly use the instructions in this standard A.1 This standard applies to chronic poisoning caused by the production or use of carbon disulfide. Carbon disulfide is mainly used in the production of sticky Rubber fiber, cellophane and rubber vulcanization and other industries. Also used for ore flotation, manufacture of carbon tetrachloride, waterproof glue, grain smoked Steamed, refined paraffin, oil and used as a solvent for dissolving fat, varnish, resin and so on. A.2 on the "long-term close occupational exposure history", generally refers to direct contact with carbon disulfide operating length of more than one year, The concentration of carbon disulfide in the air of the workshop is several times higher than the maximum allowable concentration (10mg/m3) of the national standard, and occasionally Many times. A.3 The diagnosis of mild poisoning is the starting point of positive signs of peripheral nerve damage and signs, or peripheral nerve damage performance is not Obviously, but the neuro-electromyographic examination showed positive neurogenic damage (see Standard GBZ 76 Appendix B). A.4 nerve - EMG examination of the diagnosis of this disease is important. Carbon disulfide poisoning is mainly caused by peripheral nerve axon damage This should focus on checking the distal muscles of the limbs of the EMG and distal nerve evoked potentials. Check the results and results see GBZ 76 Appendix B. A.5 carbon disulfide on the central nervous system, the early performance of brain weakness syndrome (such as headache, dizziness, insomnia, Fatigue, forgetfulness, etc.) and autonomic dysfunction (such as palpitations, sweating). Serious encephalopathy occurs when severe poisoning occurs, such as small Cerebral ataxia, Parkinson's syndrome, pyramidal tract signs (hemiplegia, pseudo-ball paralysis); or the performance of toxic psychosis; Irritability, depression, disorientation, hallucinations, delusions, or even manic or depressive psychosis. Poisonous brain Patients with brain CT or brain MRI can show brain atrophy, excluding brain degenerative diseases, vascular dementia, and other causes Of mental illness, etc., should be considered for severe poisoning. A.6 for the diagnosis of the need to rule out the cause of peripheral neuropathy of various diseases, such as furan, isoniazid, arsenic, chloropropene, Enamides, methyl n-butyl ketone, n-hexane and other poisoning, and diabetes, infectious multiple neuritis and other diseases. A.7 check the fundus to be in the large pupil after the ophthalmoscope observation, such as the discovery of retinal micro-aneurysm, the need to rule out the micro Other diseases of aneurysms such as diabetes, retinal vein occlusion, chorioretinitis, sickle cell disease, Eales disease, No disease, Costs disease, severe hypertensive retinopathy, anemia, chronic glaucoma, Leber disease, retinal mast Cell tumors and certain toxic retinopathy.

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