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Standard ID | Contents [version] | USD | STEP2 | [PDF] delivered in | Standard Title (Description) | Status | PDF |
GBZ 15-2024 | English | 139 |
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Diagnostic standard for occupational acute nitrogen oxides poisoning
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GBZ 15-2024
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GBZ 15-2002 | English | 169 |
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Diagnostic Criteria of Occupational Acute Nitrogen Oxides Poisoning
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GBZ 15-2002
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PDF similar to GBZ15-2024
Standard similar to GBZ15-2024 GBZ 57 GBZ 20 GBZ 49 GBZ 10 GBZ 6 GB/T 15
Basic data Standard ID | GBZ 15-2024 (GBZ15-2024) | Description (Translated English) | Diagnostic standard for occupational acute nitrogen oxides poisoning | Sector / Industry | National Standard | Classification of Chinese Standard | C60 | Classification of International Standard | 13.100 | Word Count Estimation | 6,668 | Date of Issue | 2024-05-09 | Date of Implementation | 2025-05-01 | Issuing agency(ies) | State Administration for Market Regulation, China National Standardization Administration | Summary | This standard specifies the diagnostic principles and diagnostic classification of occupational acute nitrogen oxide poisoning. This standard applies to the diagnosis of acute poisoning caused by occupational exposure to nitrogen oxides. |
GBZ15-2002: Diagnostic Criteria of Occupational Acute Nitrogen Oxides Poisoning---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Acute Oxides
ICS 13.100
C60
GBZ
People's Republic of China National Occupational Health Standards
Diagnostic criteria for occupational acute nitrogen oxide poisoning
Released in.2002-04-08
2002-06-01 Implementation
Issued by the Ministry of Health of the People's Republic of China
Foreword
Article 6.1 of this standard is recommended and the remainder is mandatory.
According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard
GB 7801-1987 inconsistent with this standard, the standard shall prevail.
Nitrogen oxides are a kind of irritating gas that is widely found in the chemical production process. In contact with nitrogen oxides for professional activities
, Can cause acute poisoning. In order to protect the health of contacts, prevention and treatment of acute nitrogen oxide poisoning, better improve the standard,
GB 7801-1987 was revised according to the latest developments in clinical studies.
The revised new standard not only links with GBZ 73, but also highlights the characteristics of acute nitrogen oxide poisoning, in the original standard
Based on the increase in the contents of acute respiratory distress syndrome, so that the classification of diagnosis is more clear, reasonable and easy to use.
Appendix A to this standard is an informative appendix and Appendix B is a normative appendix.
This standard is proposed and centralized by the Ministry of Health of the People's Republic of China.
This standard is drafted by Jilin Chemical Industry and Workers' Hospital, Shenyang Institute of Occupational Disease Prevention and Control, to participate in the drafting of the unit
There are Shanghai Chemical Industry Occupational Disease Prevention and Control Hospital, Dalian Chemical Industry Company Staff Hospital, Shanxi Medical University Affiliated Hospital, Bengbu Medical
College Affiliated Hospital, Shandong Provincial Hospital Occupational Diseases, Jilin Medical College.
This standard is interpreted by the Ministry of Health of the People's Republic of China.
Diagnostic criteria for occupational acute nitrogen oxide poisoning
Occupational acute nitrogen oxide poisoning refers to the occupational activity, short-term inhalation of a large number of nitrogen oxide gas, caused by
To the respiratory system of the main systemic diseases.
1 Scope
This standard specifies the diagnostic criteria and principles of occupational acute nitrogen poisoning.
This standard applies to occupational acute nitrogen poisoning, non-occupational acute nitrogen poisoning can also refer to the implementation.
2 normative reference documents
The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document
All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however,
The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest
The version applies to this standard.
GBZ 73 Occupational acute chemicals - Criteria for the diagnosis of toxic respiratory diseases
Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 16180
3 diagnostic principles
According to the short-term inhalation of a larger number of oxynitride occupational history, the clinical manifestations of respiratory damage and chest X-ray
Signs, combined with blood gas analysis and on-site labor hygiene survey data, comprehensive analysis, and exclude other causes of similar diseases
Disease, can be diagnosed.
4 stimulus response
There was a transient chest tightness, cough and other symptoms, no positive signs of the lungs, chest X-ray examination showed no abnormal performance.
5 Diagnostic and grading standards
5.1 mild poisoning
Chest, cough and other symptoms, the lungs are scattered in the dry rales; chest X-ray signs. enhanced lung texture, with edge model
Paste; meet the acute tracheal - bronchitis or bronchial inflammation.
5.2 moderate poisoning
Chest tightness increased, increased cough, difficulty breathing, expectoration or hemoptysis sputum and other symptoms; signs of mild cyanosis, lungs can be
Smell and dry, wet rales; chest X-ray signs. lung field through the brightness reduction, increased lung texture, disorder, fuzzy was reticular
Results, and Rhithers.s. Rhithers.s. Rhithers.s. Rhithers.s. Rhither wanteds. Finds.omengesomenigma rounds.omenclupulates Rhithers. Rhithers. Rh '
Blood gas analysis often mild to moderate hypoxemia.
5.3 severe poisoning
Has one of the following.
a) obvious difficulty breathing, severe cough, slightly white or pink foam sputum, obvious cyanosis, two lungs full of wet
Sexual rales; chest X-ray signs. the two lungs have the size of the edge of the blurred patchy or cloud-like shadow, there
Can be combined into a large shadow, in line with alveolar pulmonary edema. Blood gas analysis often severe hypoxemia.
b) acute respiratory distress syndrome;
c) with a heavier degree of pneumothorax or mediastinal emphysema;
d) suffocation.
6 Principles of handling
6.1 Principles of treatment
6.1.1 Field processing
Rapid, safe from the poisoning scene, lying, warm, to avoid activities, immediately oxygen; and given symptomatic treatment.
6.1.2 to stimulate the reaction, should observe 24-72 hours, the observation period should be strictly limited activities, bed rest, keep quiet,
And given symptomatic treatment.
6.1.3 to keep the airway open
To give inhalation, bronchial antispasmodic agents, to defoamers (such as dimethyl silicone oil), if necessary, to give tracheotomy.
6.1.4 early, adequate, short-range application of glucocorticoid.
6.1.5 reasonable oxygen therapy.
6.1.6 prevention and control of infection, prevention and treatment of complications, maintenance of water, electrolyte, acid-base balance.
6.2 Other treatments
Acute light and moderate poisoning can be restored after the original work; severe poisoning patients with disease recovery, should be transferred from the stimulus
Gas operation. For identification of labor capacity, according to GB/T 16180 treatment.
7 Correctly use the instructions in this standard
See Appendix A (informative).
Appendix A
(Informative)
Correctly use the instructions in this standard
A.1 The disease to the main respiratory damage, so the respiratory symptoms, signs and chest X-ray signs as the main diagnostic basis.
Such as clinical manifestations and chest X-ray performance does not match, according to the more serious indicators to make classification diagnosis.
A.2 The disease is characterized by a long incubation period, prone to delayed pulmonary edema, so close contact should be closely medical observation.
A.3 The disease in the late acute poisoning, prone to delayed obstructive bronchiolitis, should pay attention. Mainly as follows.
Pulmonary edema basically recovered about 2 weeks, and the occurrence of cough, chest tightness and progressive respiratory distress and other symptoms, signs of significant cyanosis,
Two lungs can be heard and dry rales and/or wet rales; a few cases inhaled nitrogen oxides, no obvious acute poisoning symptoms,
2 weeks after the occurrence of the above lesions. Chest X-ray signs. two lungs full of miliary shadows. On the occurrence of delayed obstructive bronchioles
Yan, should be given glucocorticoid treatment.
A.4 Diagnosis of Acute Respiratory Distress Syndrome GBZ 73.
A.5 blood gas analysis as a reference index for diagnostic grading, is of great significance, with reference to GBZ 73.
A.6 reasonable oxygen therapy in the treatment of nitrogen oxide poisoning is of great significance, with reference to GBZ 73.
A.7 The key to the treatment of this disease is the active prevention and treatment of pulmonary edema, early, adequate, short-term application of glucocorticoids,
Scopolamine drugs such as 654-2.
A.8 aerosol inhalation is one of the effective ways to treat acute toxic respiratory diseases, depending on the condition 2-3 times a day,
Ultrasonic atomization inhalation.
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