GBZ39-2016 English PDF

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GBZ39-2016: Diagnosis of occupational acute 1, 2-dichloroethane poisoning
Status: Valid

GBZ39: Historical versions

Standard ID	USD	BUY PDF	Lead-Days	Standard Title (Description)	Status
GBZ 39-2016	109	Add to Cart	3 days	Diagnosis of occupational acute 1, 2-dichloroethane poisoning	Valid
GBZ 39-2002	239	Add to Cart	3 days	Diagnostic Criteria of Occupational Acute 1, 2 -- Dichloroethane Poisoning	Obsolete

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Basic data

Standard ID: GBZ 39-2016 (GBZ39-2016)
Description (Translated English): Diagnosis of occupational acute 1, 2-dichloroethane poisoning
Sector / Industry: National Standard
Classification of Chinese Standard: C60
Classification of International Standard: 13.100
Word Count Estimation: 5,580
Date of Issue: 2016-08-23
Date of Implementation: 2017-02-01
Older Standard (superseded by this standard): GBZ 39-2002
Quoted Standard: GB/T 16180; GBZ 76
Regulation (derived from): State-Health-Announcement (2016)14
Issuing agency(ies): General Administration of Quality Supervision, Inspection and Quarantine of the People's Republic of China
Summary: This standard specifies the principles of diagnosis and treatment of occupational acute 1, 2-dichloroethane poisoning. This standard applies to occupational activities in contact with 1, 2 - dichloroethane caused by the diagnosis and treatment of acute poisoning. Subacute poisoning diagnosis and treatment can refer to the use.

GBZ39-2002: Diagnostic Criteria of Occupational Acute 1, 2 -- Dichloroethane Poisoning

---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic Criteria of Occupational Acute 1, 2.Dichloroethane Poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational acute 1,2 - dichloroethane poisoning Diagnostic Criteria of Occupational Acute L, 2-Dichloroethane Poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China

Foreword

Article 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 11506-1989 inconsistent with this standard, subject to this standard. In the exposure to 1,2-dichloroethane (hereinafter referred to as dichloroethane) in the professional activities can cause acute poisoning (including sub Acute poisoning), in order to protect the health of contacts, in 1989 the country promulgated GB ll506-1989 "occupational acute 1,2 Dichloroethane poisoning diagnostic criteria and treatment principles ". This standard in the diagnostic system and GBZ 71 and GBZ 76 phase convergence. Combined with recent advances in research, highlighting the impact of dichloroethane on the central nervous system, especially in the diagnostic criteria Poisoning caused by poisoning encephalopathy, and in the article on the revised. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. This standard by the Guangdong Provincial Occupational Disease Prevention and Control Hospital, Shenzhen City Health and Epidemic Prevention Station is responsible for drafting, Bao'an District of Shenzhen City Health and Epidemic Prevention Station, Zhuhai City Health and Epidemic Prevention Station to participate in the drafting. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational acute 1,2 - dichloroethane poisoning Occupational acute dichloroethane poisoning is in the occupational activity, short-term exposure to higher concentrations of dichloroethane caused by the central god The systemic damage caused by systemic diseases, may have liver and kidney damage.

1 Scope

This standard specifies the diagnostic criteria and principles of occupational acute dichloroethane poisoning. This standard applies to occupational activity in contact with dichloroethane caused by acute and subacute poisoning diagnosis and treatment.

2 normative reference documents

The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest The version applies to this standard. Diagnostic criteria for occupational toxic liver disease in GBZ 59 GBZ 76 Occupational acute chemical poisoning Nervous system disease diagnostic criteria Diagnostic criteria for occupational acute kidney disease Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 16180

3 diagnostic principles

According to the occupational history of exposure to higher concentrations of dichloroethane and the clinical manifestations of central nervous system damage Combined with the scene of labor hygiene investigation, comprehensive analysis, excluding other causes caused by similar diseases can be diagnosed.

4 contact reaction

Short-term exposure to higher concentrations of dichloroethane, the emergence of dizziness, headache, fatigue and other central nervous system symptoms, with nausea, Vomiting or eye and upper respiratory tract irritation symptoms, from the contact after a short time to disappear.

5 Diagnostic and grading standards

5.1 mild poisoning In addition to the above symptoms, the following manifestation appears. a) gait staggering; b) mild disturbance of consciousness, such as confusion, lethargy state, hazy state; c) mildly toxic liver disease; d) mildly toxic nephropathy. 5.2 severe poisoning One of the following occurrences. a) moderate or severe disturbance of consciousness; b) epilepsy seizures; c) brain focal lesions, such as cerebellar ataxia; d) moderate or severe toxic liver disease.

6 Principles of handling

6.1 Principles of treatment 6.1.l on-site treatment. the poison should be quickly removed from the scene, moved to fresh air, replaced by contaminated clothing, washed Contaminated skin, warm, and closely observed. 6.1.2 Respondents should be closely observed and given symptomatic treatment. 6.1.3 to focus on the prevention and treatment of toxic encephalopathy, active treatment of cerebral edema, reduce intracranial pressure. 6.1.4 no special effects antidote, treatment principles and care and neurology, the same medical. 6.2 Other treatments Mild poisoning can be restored after the original work. Severe poisoning should be transferred after the recovery of dichloroethane operations, the need for labor capacity Identification by GB/T 16180 treatment.

7 Correctly use the instructions in this standard

See Appendix A (informative).

Appendix A

(Informative) Correctly use the instructions in this standard Al This standard applies to occupational exposure to dichloroethane-induced acute and subacute poisoning. Contact in non-professional activities Dichloroethane caused by poisoning, can also refer to the use of this standard. Dichloroethane commonly used as chemical synthesis of raw materials, industrial solvents, degreasing agents, metal cleaning agents and adhesives. A.2 Acute and subacute dichloroethane poisoning is mainly manifested as central nervous system damage. Especially subacute poisoning is nearly a decade To the main form of the disease, it is seen in a long time inhalation of higher concentrations of poisoning in patients. Its characteristics. a longer incubation period, A few days to several days; onset concealment, the condition can suddenly deteriorate; clinical manifestations of toxic encephalopathy, highlighting the performance of brain water Swelling, some severe poisoning may have brain lesions damaged performance, such as cerebellar ataxia and so on. Consciousness disorder Light, medium and severe Are divided according to Appendix D in GBZ 76. A.3 acute diclofenac poisoning encephalopathy, cerebral edema can be sustained for about two weeks, and can be repeated or sudden increase. Treatment should be To prevent brain edema, reduce intracranial pressure-based, stressed that "close observation, early detection, timely treatment, to prevent repeated." And Treatment time should not be less than two weeks. A.4 acute dichloroethane poisoning, the clinical observation shows liver and kidney damage, but more common in oral poisoning. Occupational poisoning Serious liver damage, kidney damage is very rare, especially the kidney. Symptoms of toxic liver disease and toxic nephropathy See GBZ 59 And GBZ 79. ......

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