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GBZ28-2010 English PDF

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GBZ28-2010: Diagnostic criteria of occupational acute nickel carbonyl poisoning
Status: Valid

GBZ28: Historical versions

Standard IDUSDBUY PDFLead-DaysStandard Title (Description)Status
GBZ 28-2010139 Add to Cart 3 days Diagnostic criteria of occupational acute nickel carbonyl poisoning Valid
GBZ 28-2002199 Add to Cart 3 days Diagnostic criteria of occupational acute nickel carbonyl poisoning Obsolete

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Basic data

Standard ID: GBZ 28-2010 (GBZ28-2010)
Description (Translated English): Diagnostic criteria of occupational acute nickel carbonyl poisoning
Sector / Industry: National Standard
Classification of Chinese Standard: C60
Classification of International Standard: 13.100
Word Count Estimation: 6,670
Date of Issue: 2010-03-10
Date of Implementation: 2010-10-01
Older Standard (superseded by this standard): GBZ 28-2002
Quoted Standard: GBZ 73; GB/T 16180; WS/T 44; WS/T 45
Regulation (derived from): ?Health-Communication (2010) 6
Issuing agency(ies): Ministry of Health of the People's Republic of China
Summary: This standard specifies the principles of diagnosis and treatment of occupational acute nickel carbonyl poisoning. This standard applies to the diagnosis and treatment of occupational acute nickel carbonyl poisoning. Other metal carbonyl (such as carbonyl iron, cobalt carbonyl, etc.) in the diagnosis and treatment of acute poisoning and non- occupational acute nickel carbonyl poisoning can also use and reference.

GBZ28-2002: Diagnostic criteria of occupational acute nickel carbonyl poisoning

---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.
Diagnostic criteria of occupational acute nickel carbonyl poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational acute carbonyl nickel poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China

Foreword

Article 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 8786-1988 inconsistent with this standard, the standard shall prevail. Carbonyl nickel is commonly used in metal smelting, purification, catalysis and other industries. In contact with carbonyl nickel in the professional activities, often can cause Exposure to acute poisoning. In order to protect the health of contacts, prevention and control of carbonyl nickel poisoning, according to recent advances in clinical research work, The standard GB 8786-1988 was revised. The revised standard is not only in line with the GBZ 73 standard, but also highlights the characteristics of acute carbonyl nickel poisoning itself, in the original Diagnostic criteria based on the addition and modification. So that the classification of diagnosis more clear, full management, easy to apply. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. The revised version of this standard by the Suzhou University School of Medicine, Dalian City Institute of Labor and Health is responsible for drafting, nuclear industry 857 Plant, nuclear industry 8, Shanghai Sanstei Hospital to participate in the drafting. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational acute carbonyl nickel poisoning Occupational acute carbonyl nickel poisoning is caused by exposure to a greater amount of carbonyl nickel in a short period of time in occupational activities. Respiratory system damage as the main manifestation of systemic disease.

1 Scope

This standard specifies the diagnostic criteria and principles of occupational acute carbonyl nickel poisoning. This standard is applicable to the diagnosis and treatment of occupational acute carbonyl nickel poisoning. Non-occupational acute carbonyl nickel poisoning diagnosis, Also refer to this standard.

2 normative reference documents

The terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest The version applies to this standard. GBZ 73 Occupational acute chemicals - Criteria for the diagnosis of toxic respiratory diseases Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 16180

3 diagnostic principles

According to the short-term exposure to a large number of carbonyl nickel occupational history, respiratory system damage and chest X-ray performance, Combined with blood gas analysis, reference to on-site labor hygiene survey, comprehensive analysis, excluding other causes of similar diseases, Off.

4 stimulus response

There is a transient upper respiratory tract irritation symptoms, no positive signs of the lungs, chest X-ray film without abnormal performance.

5 Diagnostic and grading standards

5.1 mild poisoning Dizziness, headache, fatigue, drowsiness, chest tightness, throat, nausea, loss of appetite and other symptoms; physical examination visible conjunctiva and Pharyngeal mild hyperemia, two lungs smell and scattered dry, wet rales; chest X-ray examination normal or show two lungs increased, increased Rough, blurred edges. The above performance in line with acute bronchitis or bronchial inflammation. 5.2 moderate poisoning One of the following. a) cough, sputum, shortness of breath, chest tightness, may have sputum bloody or mild cyanosis; two lungs have obvious dry, wet rales; Chest X-ray examination showed enhanced lung texture, blurred edges, middle and lower lung field spotted or patchy shadows. Above performance Complies with acute bronchial pneumonia; b) cough, sputum, shortness of breath heavier; breath sounds reduced; chest X-ray examination showed hilar shadow blur increases, two Lung scattered in the small dotted shadows and reticular shadows, lung field through the brightness decreased. The above performance in line with acute interstitial pulmonary edema. Blood gas analysis often mild to moderate hypoxemia. 5.3 severe poisoning One of the following. a) cough a lot of white or pink foam sputum, obvious difficulty breathing, cyanosis, diffuse wet rales of the lungs; Chest X-ray examination showed two different sizes of the lung field, the edge of the flaky flake or cloud-like shadow, and sometimes can be merged into large Or butterfly-like distribution. The above performance is consistent with alveolar pulmonary edema; b) acute respiratory distress syndrome. Blood gas analysis often severe hypoxemia.

6 Principles of handling

6.1 Principles of treatment 6.1.1 immediately from the poisoning scene, take off the contaminated clothing. Clean contaminated skin and hair, stay in bed and keep it safe Static. Close observation and given symptomatic treatment. 6.1.2 correct hypoxia to give oxygen inhalation and keep the airway open. 6.1.3 prevention and treatment of pulmonary edema should be early, adequate, short-term application of glucocorticoids, control the amount of liquid input. You can apply defoaming (Dimethyl silicone oil aerosol). 6.1.4 to prevent infection, prevention and treatment of complications, to maintain electrolyte balance. 6.1.5 severe poisoning may be diethyldithiocarbamate (dithiocarb) orally, each 0.5g, 4 times a day, And at the same time taking the same amount of sodium bicarbonate, according to the disease decided with the number of days, generally continuous medication 3-7 days. Can also be used to smoke suction In. 6.2 Other treatments Mild, moderate poisoning patients can be restored after the original work. Severe poisoning patients after treatment is still obvious symptoms should be discretion Arranged to rest, and transferred from carbonyl nickel operations. For identification of labor capacity, according to GB/T 16180 treatment.

7 Correctly use the instructions in this standard

See Appendix A (informative).

Appendix A

(Informative) Correctly use the instructions in this standard A.1 This standard applies to acute carbonyl nickel poisoning. Other carbonyl metals such as carbonyl iron, carbonyl cobalt acute poisoning can refer to use. A.2 The diagnostic grade of this standard is based on the degree of damage to the respiratory system, the stimulus response occurs after exposure to carbonyl nickel A transient response, has not yet reached the degree of poisoning, in order to closely observe the disease development, easy to deal with, included in the classification standard Quasi but not acute poisoning. A.3 contact with carbonyl nickel workers suspected acute poisoning may have to be closely clinical observation. Observation time of not less than 48 hour. A.4 acute carbonyl nickel poisoning pulmonary edema, resulting in hypoxia, blood gas analysis PaO2 determination can understand the degree of hypoxia, But the correct judgment of the disease should be combined with clinical and dynamic analysis of data comprehensive analysis. A.5 severe acute poisoning often due to hypoxia Erzhi ECG, liver, kidney function changes. These changes can often be corrected with hypoxia Is recovered, and is not included in the diagnostic terms. A.6 Acute respiratory distress syndrome (ARDS) diagnosis GBZ 73. A.7 in order to master the overall condition of poisoning, severe poisoning patients in addition to chest X-ray examination, according to the disease choose to check the heart Electricity, liver, kidney function. Respiratory system to alleviate the acute symptoms of patients with postoperative patients need to be pulmonary ventilation function determination. A.8 Early application of sodium diethyldithiocarbamate has a preventive effect on toxic pulmonary edema caused by carbonyl nickel.
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