GBZ29-2011 English PDFUS$139.00 · In stock
Delivery: <= 3 days. True-PDF full-copy in English will be manually translated and delivered via email. GBZ29-2011: Diagnosis of occupational acute phosgene poisoning Status: Valid GBZ29: Historical versions
Basic dataStandard ID: GBZ 29-2011 (GBZ29-2011)Description (Translated English): Diagnosis of occupational acute phosgene poisoning Sector / Industry: National Standard Classification of Chinese Standard: C60 Classification of International Standard: 13.100 Word Count Estimation: 6,637 Date of Issue: 2011-04-21 Date of Implementation: 2011-11-01 Older Standard (superseded by this standard): GBZ 29-2002 Quoted Standard: GB/T 16180; GBZ 73; GBZ 74 Regulation (derived from): ?Health-Communication (2011) 7 Issuing agency(ies): Ministry of Health of the People's Republic of China Summary: This standard specifies the principles of diagnosis of occupational acute phosgene poisoning, diagnosis and classification and treatment principles. This standard applies to occupational acute phosgene poisoning diagnosis and treatment. Acute occupational poisoning and double phosgene poisoning may refer to the use of triphosgene. Non- occupational acute poisoning may also refer to the use of phosgene. GBZ29-2002: Diagnostic criteria of occupational acute phosgene poisoning---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.Diagnostic criteria of occupational acute phosgene poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational acute phosgene poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China ForewordArticle 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 8787-1988 inconsistent with this standard, the standard shall prevail. In contact with phosgene professional activities can cause acute poisoning, in order to standardize the diagnosis of acute phosgene poisoning, to protect the contact body Health, has released GB 8787-1988. The revised standard is not only in line with the GBZ 73, but also highlights the characteristics of phosgene poisoning, based on the original standard increased Acute respiratory distress syndrome (ARDS) diagnosis and so on, so that the classification of diagnosis is more clear, reasonable and easy to use. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. This standard by the Shanghai Yangpu District Central Hospital, Shanghai Wusong Chemical Plant is responsible for drafting, to participate in the drafting of the units are North Third Hospital of Beijing University, Shanghai Chemical Industry Supervision Institute, Zhejiang Center for Disease Control and Prevention, Shanghai Occupational Disease Prevention and Control Hospital, Guang East Province Occupational Disease Prevention and Control Hospital, Shantou City Occupational Disease Prevention and Control, Gansu Silver Chemical Industry Company workers hospital. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational acute phosgene poisoning Occupational acute phosgene poisoning is in the short-term occupational activities inhalation caused by a large number of phosgene caused by acute respiratory failure Harm the main systemic disease. Prone to pulmonary edema.1 ScopeThis standard specifies the diagnostic criteria and principles of occupational acute phosgene poisoning. This standard applies to the diagnosis and treatment of occupational acute phosgene poisoning. Non-occupational acute phosgene poisoning can be used.2 normative reference documentsThe terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest The version applies to this standard. GBZ 73 Occupational acute chemicals - Criteria for the diagnosis of toxic respiratory diseases Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 161803 diagnostic principlesAccording to a clear short-term exposure to phosgene occupational history, acute respiratory system damage to the clinical symptoms, signs, chest X-ray Performance, combined with blood gas analysis and other checks, refer to the scene of labor hygiene survey data, comprehensive analysis, excluding other causes Caused by similar diseases, can be diagnosed.4 stimulus responseThere is a transient eye and upper respiratory tract mucosal irritation symptoms, no positive signs of the lungs, chest X-ray performance without exception change.5 Diagnostic and grading standards5.1 mild poisoning Cough, shortness of breath, chest tightness or chest pain, the lungs may have scattered dry, wet rales. X-ray chest radiograph showed enhanced lung texture Or with edges blurred. The above performance in line with bronchitis or bronchial inflammation. 5.2 moderate poisoning One of the following. a) chest tightness, shortness of breath, cough, sputum, etc., may have sputum bloody, often accompanied by mild cyanosis, lungs appear dry, wet Rales, chest X-ray performance for the two, the lower lung field visible spot or small patchy shadow. The above performance is consistent Acute bronchial pneumonia; b) chest tightness, shortness of breath, cough, sputum more serious, the two lungs breath sounds reduced, no obvious rales, chest X-ray Manifested as increased lung texture, hilar shadow widened, realm is unclear, the two lungs scattered in the dotted shadows and reticular shadows, Lung field transparency reduced, often seen horizontal crack thickening, and sometimes visible bronchial cuff sign or grams of B line. the above Performance consistent with acute interstitial pulmonary edema. Blood gas analysis is often mild or moderate hypoxemia. 5.3 severe poisoning One of the following. a) obvious difficulty breathing, cyanosis, frequent cough, slightly white or pink foam sputum, the lungs have a wide range of wet rales, Chest X-ray performance for the two lungs have different sizes, the edge of the small flake, cloud flocculent or cotton-like shadow, Sometimes can be integrated into a large shadow or butterfly-shaped distribution, blood gas analysis showed PaO2/FiO2 ≤ 40kPa (300 mmHg). The above performance in line with diffuse alveolar pulmonary edema or central alveolar pulmonary edema; b) the above situation is more serious, respiratory rate ( > 28 times/min) or (and) respiratory distress, chest X-ray shows two The lungs were fused with large lamellae, and blood gas analysis showed PaO2/FiO2 ≤ 26.7 kPa (200 mmHg). Above performance Consistent with acute respiratory distress syndrome; c) suffocation; d) complicated with pneumothorax, mediastinal emphysema; e) severe myocardial damage; f) shock g) coma.6 Principles of handling6.1 Principles of treatment 6.1.1 Inhalation of phosgene should be quickly from the scene to the fresh air, immediately remove the contaminated clothing, body surface with a liquid The parts of the phosgene are washed thoroughly with water. Keep quiet, absolutely bed rest, keep warm. Early oxygen, given drug fog Inhalation, with bronchial spasmoditis, antitussive, sedative and other symptomatic treatment. At least close observation of 48h, pay attention to changes in condition. 6.1.2 Prevention and treatment of pulmonary edema. Early, adequate, short-range application of glucocorticoid, control of liquid input. You can use defoamers Such as dimethyl silicone oil aerosol inhalation, pay attention to keep the airway patency. Reasonable oxygenation; Inhalation oxygen concentration (FiO2) should not exceed 60%. 6.1.3 Treatment of acute respiratory distress syndrome. refer to GB 73 of the relevant content. Other first aid treatment and prevention of complications with the same Principles of Internal Medicine Treatment. 6.2 Other treatments Acute poisoning patients cured, can restore the original work. Severe poisoning patients such as X-ray, blood gas analysis or lung function Such as the determination of abnormal performance, should be transferred from the stimulating gas operations. Those who need to identify labor capacity, with reference to GB/T 16180.7 Correctly use the instructions in this standardSee Appendix A (informative).Appendix A(Informative) Correctly use the instructions in this standard A.1 acute phosgene poisoning to the main respiratory damage, which is characterized by frequent onset of pulmonary edema, the occurrence of lung water Swollen incubation period of up to 48h, during which patients can be no obvious clinical symptoms, signs, should be noted. A.2 The disease may be accompanied by other system symptoms such as dizziness, fatigue, nausea, increased total white blood cells, fever, etc. Poisoning severity is not exactly the same, so the poisoning diagnostic grading standards are still respiratory symptoms, signs and chest X-ray table Is the main basis. Blood gas analysis is an important reference for severity of illness. A.3 severe poisoning can be complicated by other organ damage, such as shock, myocardial damage, coma, etc., the first aid treatment with internal medicine. A.4 hypoxemia blood gas analysis grading as follows. mild hypoxemia; PaO2 ≤ 10.7kPa (80mmHg). Medium low Oxygen; PaO2 ≤ 8kPa (60mmHg). Severe hypoxemia; PaO2 ≤ 5.3 kPa (40 mmHg). This standard is heavy Degree of poisoning caused by acute respiratory distress syndrome diagnostic indicators, with reference to foreign data and to determine the PaO2/FiO2 ≤ 40kPa (300mmHg) as a major indicator of toxic diffuse alveolar pulmonary edema, PaO2/FiO2 ≤ 26.7kPa (200 mmHg) As a diagnostic indicator of ARDS. Blood gas analysis should be combined with clinical manifestations for dynamic observation. A.5 stimulating reactions and poisoning commonly used drugs ultrasonic atomization inhalation. Recipe. dexamethasone 5mg; aminophylline 0.25g; celebration Moxycin 80,000, the amount of saline, the effect is better. Poisoning patients should keep the airway patency, if necessary, to give defoaming net inhalation, etc. or the implementation of tracheal intubation or tracheotomy Surgery A.6 acute respiratory distress syndrome prevention and treatment should be emphasized in the comprehensive treatment. refer to GBZ 73 related content. A.7 acute phosgene after poisoning, can occur in the recovery of occlusive bronchiolitis, usually in the acute phase of symptoms after 2 weeks Left and right, should cause attention, so as not to misdiagnosis. ......Tips & Frequently Asked Questions:Question 1: How long will the true-PDF of GBZ29-2011_English be delivered?Answer: Upon your order, we will start to translate GBZ29-2011_English as soon as possible, and keep you informed of the progress. The lead time is typically 1 ~ 3 working days. The lengthier the document the longer the lead time.Question 2: Can I share the purchased PDF of GBZ29-2011_English with my colleagues?Answer: Yes. 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