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Delivery: <= 3 days. True-PDF full-copy in English will be manually translated and delivered via email. GBZ13-2016: Diagnosis of occupational acute acrylonitrile poisoning Status: Valid GBZ13: Historical versions
Basic dataStandard ID: GBZ 13-2016 (GBZ13-2016)Description (Translated English): Diagnosis of occupational acute acrylonitrile poisoning Sector / Industry: National Standard Classification of Chinese Standard: C60 Classification of International Standard: 13.100 Word Count Estimation: 5,560 Date of Issue: 2016-08-23 Date of Implementation: 2017-02-01 Older Standard (superseded by this standard): GBZ 13-2002 Quoted Standard: GB/T 16180; GBZ 76; GBZ 78 Regulation (derived from): State-Health-Announcement (2016)14 Issuing agency(ies): General Administration of Quality Supervision, Inspection and Quarantine of the People's Republic of China Summary: This standard specifies the diagnostic principles of occupational acute acrylonitrile poisoning, diagnostic grading and treatment principles. This standard applies to the diagnosis and treatment of occupational acute acrylonitrile poisoning. GBZ13-2002: Diagnostic Criteria of Occupational Acute Acrylonitrile Poisoning---This is a DRAFT version for illustration, not a final translation. Full copy of true-PDF in English version (including equations, symbols, images, flow-chart, tables, and figures etc.) will be manually/carefully translated upon your order.Diagnostic Criteria of Occupational Acute Acrylonitrile Poisoning ICS 13.100 C60 GBZ People's Republic of China National Occupational Health Standards Diagnostic criteria for occupational acute acrylonitrile poisoning Released in.2002-04-08 2002-06-01 Implementation Issued by the Ministry of Health of the People's Republic of China ForewordArticle 6.1 of this standard is recommended and the remainder is mandatory. According to the "People's Republic of China Occupational Disease Prevention Law" to develop this standard. Since the implementation of the standard date, the original standard GB 7799-1987 inconsistent with this standard, subject to this standard. Acrylic (ACN) is an important monomer for synthetic chemicals and is used in a wide range of applications. May occur due to exposure to a greater amount of acrylonitrile Acute poisoning. In order to effectively control acute acrylonitrile poisoning, GB 7799-1987 has been promulgated. The original standard promulgated 11 years, continue to have new cases reported that the clinical experience is more abundant, toxicology research also has a certain , And therefore need to be revised. The revised standard highlights the occupational acute acrylonitrile poisoning is the central nervous system Damage as the main performance of the systemic disease, and may be heart, liver, lung and other damage, so that the standard diagnostic criteria more clearly Indeed, easy to operate. The appendix adds a description of the relationship between the general criteria for occupational acute chemical poisoning, Clear and reasonable. Appendix A to this standard is an informative appendix. This standard is proposed and centralized by the Ministry of Health of the People's Republic of China. This standard by the Lanzhou Chemical Industry Company workers hospital, Fudan University Jinshan Hospital is responsible for drafting. Ningbo City Health Prevention Epidemic station, Anqing Petrochemical Plant Hospital, Urumqi Petrochemical Plant Hospital, Ningbo Zhenhai District Health and Epidemic Prevention Station to participate in the drafting. This standard is interpreted by the Ministry of Health of the People's Republic of China. Diagnostic criteria for occupational acute acrylonitrile poisoning Occupational acute acrylonitrile poisoning refers to the occupational activity, a short period of time exposure to a large number of acrylic eyes caused by the central god The systemic damage caused by systemic diseases, may be associated with heart, liver, lung and other organ damage.1 ScopeThis standard specifies the diagnostic criteria and principles of occupational acute acrylonitrile poisoning. This standard applies to the occupational exposure to acrylonitrile caused by the diagnosis and treatment of acute poisoning. Non - occupational acute acrylonitrile Poisoning can refer to use.2 normative reference documentsThe terms of the following documents are hereby incorporated by reference into this standard. Any date that references the date of the document All subsequent amendments (excluding corrigenda) or revisions do not apply to this standard, however, The parties to the agreement are able to use the latest version of these documents. Those who do not mind the date of the reference file, its latest The version applies to this standard. GBZ 18 diagnostic criteria for occupational dermatosis (general) Diagnostic criteria for occupational toxic liver disease GBZ 73 Occupational acute chemicals - Criteria for the diagnosis of toxic respiratory diseases Diagnostic criteria for occupational acute chemical toxic heart disease GBZ 74 GBZ 76 Occupational acute chemical poisoning Nervous system disease diagnostic criteria Identification of Occupational Injury and Occupational Disease Disability in Staff and Workers GB/T 161803 diagnostic principlesAccording to a short period of time exposure to a large number of acrylonitrile occupational history, the central nervous system damage as the main clinical manifestations, combined Site labor hygiene survey results of a comprehensive analysis, excluding other causes of similar diseases, can be diagnosed.4 contact reactionHeadache, dizziness, fatigue, throat, conjunctiva and nasopharyngeal hyperemia and other performance, from the contact after a short period of time to recover.5 Diagnostic and grading standards5.1 mild poisoning Headache, dizziness, upper abdominal discomfort, nausea, vomiting, numbness, chest tightness, difficulty breathing, tendon hyperreflexia Intravenous, lethargy or blurred state, may have elevated serum transaminase, ECG or myocardial abnormalities. 5.2 severe poisoning On the basis of mild poisoning, the following person appears. a) epilepsy seizures; b) coma; c) Pulmonary edema.6 Principles of handling6.1 Principles of treatment 6.1.l quickly out of the scene, take off contaminated clothing, skin contaminated parts thoroughly washed with water. 6.1.2 contact with the respondent should be closely observed, the symptoms of severe symptomatic treatment. Mild poisoning can be intravenous sodium thiosulfate; Severe poisoning using methemoglobin formation agent and sodium thiosulfate, sodium thiosulfate according to the disease can be reused. 6.1.3 to oxygen, according to the disease using hyperbaric oxygen therapy. 6.1.4 symptomatic treatment, such as cerebral edema can be applied glucocorticoid and dehydration, diuresis and other treatment. 6.2 Other treatments 6.2.l mild poisoning by the appropriate rest after treatment can restore the original work. 6.2.2 severe poisoning such as neurological symptoms, signs of incomplete recovery, should be transferred from the original job, and according to the condition recovery needs Continue to rest or arrange light work. For the identification of labor capacity by GB/T 16180.7 Correctly use the instructions in this standardSee Appendix A (informative).Appendix A(Informative) Correctly use the instructions in this standard A.1 This standard applies to acute poisoning caused by exposure to a large amount of acrylonitrile in a short period of time. A.2 contact reaction is a transient reaction, does not belong to poisoning, usually within 24 hours after disengagement from recovery. A.3 poisoning latency of about 0.5 to 24 hours, so contact with the reaction of at least 24 hours. A.4 According to the central nervous system damage after poisoning based on the clinical characteristics, where there are drowsiness or blurred consciousness were classified as mild Poison, coma, convulsions were identified as severe poisoning, reference to acute chemical poisoning neurological disease diagnostic criteria. A.5 clinical see some cases can occur pulmonary edema, so that there are breathing difficulties, wet rales and other performance of the two lungs, X-ray examination There are patchy shadows, in line with alveolar pulmonary edema according to GBZ 73-2002 "Occupational acute chemical toxic respiration System disease diagnosis criteria ", also designated as severe poisoning. A.6 acute poisoning can occur mild liver damage, such as abnormal liver function; can also occur mild heart damage, manifested as ECG Figure ST segment and T wave changes, but the recovery is fast, it is mild poisoning. Severe toxic liver disease and severe toxic heart disease Clinical rare. Such as the presence of heart or liver damage, the diagnosis of grading and treatment, with reference to GBZ 59, GBZ 74. A.7 acrylonitrile on the skin, mucous membrane has a strong stimulating effect, contact with a few hours may be different degrees of skin damage; but Does not belong to the scope of acute acrylonitrile poisoning, such as the occurrence of reference to GBZ 18 for diagnosis and treatment. A.8 Description of treatment A.8.1 At present at home and abroad is still nitrite, sodium thiosulfate as acute acrylonitrile poisoning preferred detoxification drugs, efficacy reliable. Mild poisoning intravenous sodium thiosulfate 5 ~ 10g, no need to use methemoglobin formation agent; severe poisoning Can be intravenous injection of 3% sodium nitrite 5 ~ 10ml or intramuscular injection of 10% 4-dimethyl amino phenol 2ml and then intravenous sodium thiosulfate 10 ~ 15g, 30 minutes after treatment, the condition has not been reduced, can be repeated application of sodium thiosulfate 5 ~ 10g. A.8.2 active oxygen, conditional conditions can be given according to the condition of hyperbaric oxygen therapy. A.8.3 severe poisoning patients in the application of special detoxification drugs at the same time, early, short, adequate application of glucocorticoids, Help to prevent brain edema and other injuries. A.9 Urine thiocyanate determination can be used as an indication of acrylonitrile contact, only for diagnostic reference, so the determination method is not included in the standard Quasi-appendix; domestic determination of more methods, the general use of pyridine - barbituric acid colorimetric method. ......Tips & Frequently Asked Questions:Question 1: How long will the true-PDF of GBZ13-2016_English be delivered?Answer: Upon your order, we will start to translate GBZ13-2016_English as soon as possible, and keep you informed of the progress. The lead time is typically 1 ~ 3 working days. The lengthier the document the longer the lead time.Question 2: Can I share the purchased PDF of GBZ13-2016_English with my colleagues?Answer: Yes. 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